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Experiments on a spinal cord injury (SCI) model have shown that the nitric oxide (NO) causes cardiac dysfunction. The aim of the present work was to study the impact of administration of NG-nitro-L-arginine methyl ester (L-NAME), a NO-synthase inhibitor, on the systemic blood flow, cardiac function, and haemodynamics in the control and SCI rats. Rats received 30 mg/kg/day L-NAME by intraperitoneal injection for 14 days. After 21 days of the drug administration, the SCI was induced by a brief impact of a mechanical injury of the spinal cord on the the spinal cord impactor. The blood flow measurements in the left ventricle and in the femoral vessels were performed after injecting the radio-isotope, 3H-labeled microspheres, and light emission of the fluorescein isothiocyanate was used to determine the myocardial contractility indices (pressure-volume ratio, and pressure half time of the contraction). The SCI reduced the blood flow in the femoral artery by 20% and in the left ventricle by 47% in comparison to the sham-operated rats (p < 0.001). The blood flow in the femoral vein was increased by 19% (p < 0.05) in the SCI rats as compared with the control rats. L-NAME administration to the control rats did not change the blood flow in the heart and in the femoral vessels. However, this drug caused a prominent (p < 0.05) rise in the blood flow in the femoral vein and a decrease in the blood flow in the femoral artery in the SCI rats, as compared with the SCI + saline rats

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Descargar Solucionario Del Libro Calculo Integral Moises 79 ((EXCLUSIVE))

Descargar Solucionario Del Libro Calculo Integral Moises 79 ((EXCLUSIVE))

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